Cov ntaub ntawv yooj yim | |
Khoom npe | Diclofenac sodium |
Qib | qib tshuaj |
Qhov tshwm sim | Ib tug dawb los yog me ntsis yellowish crystalline hmoov |
Assay | 99% |
Txee lub neej | 4 Xyoo |
Ntim | 25kg / thawv |
Qhov xwm txheej | Khaws lub thawv kaw rau hauv qhov chaw qhuav, zoo-ventilated. |
Kev piav qhia ntawm Diclofenac sodium
Pharmaceutical Secondary qauv rau daim ntawv thov nyob rau hauv kev tswj kom zoo, muab cov chaw soj nstuam tshuaj thiab manufacturers nrog ib tug yooj yim thiab nqi-zoo lwm txoj rau kev npaj cov txheej txheem ua hauj lwm hauv tsev.
Nws yog categorized nyob rau hauv chav kawm ntawm non-steroidal anti-inflammatory tshuaj (NSAIDs). Nws qhia inflammatory, analgesic thiab antipyretic kev ua ub no. Diclofenac Sodium yog sodium ntsev daim ntawv ntawm diclofenac, ib tug benzene acetic acid derivate thiab nonsteroidal anti-inflammatory tshuaj (NSAID) nrog analgesic, antipyretic thiab anti-inflammatory kev ua.
Diclofenac sodium tau ntev tau siv los kho mob hnyav thiab mob, thiab siv tau rau ntau hom mob.
Daim ntawv thov kev kho mob txog Diclofenac sodium
Cov kev sim tshuaj kho mob tau ua kom pom qhov ua tau zoo ntawm diclofenac sodium nyob rau hauv cov nqe lus ntawm kev txo qis qhov mob me me mus rau qhov mob hnyav tom qab phais hauv cov neeg mob kho hniav lossis phais me me. Subcutaneous diclofenac sodium kuj tau txais txiaj ntsig zoo txo qis rau qhov mob neuropathic hnyav, ntsig txog mob qog noj ntshav lossis tsis. Diclofenac sodium feem ntau tau txais txiaj ntsig zoo hauv kev sim tshuaj, nrog rau kev txhaj tshuaj-qhov tshuaj tiv thaiv ntawm cov xwm txheej tshwm sim feem ntau. Diclofenac sodium yog qhia rau kev kho mob rheumatoid mob caj dab, osteoarthritis, thiab ankylosing spondylitis.
Mechanisms ntawm kev txiav txim txog Diclofenac sodium
Putative mechanisms ntawm kev txiav txim ntawm diclofenac yuav muaj xws li inhibition ntawm leukotriene synthesis, inhibition ntawm phospholipase A2, modulation ntawm dawb arachidonic acid theem, stimulation ntawm adenosine triphosphate-sensitive poov tshuaj raws ntawm L-arginine-nitric oxide-cyclic guanosine monophosphate mediaway thiab central kab. neuropathic mechanisms. Lwm cov txheej txheem tshwm sim ntawm kev ua haujlwm yuav suav nrog inhibition ntawm peroxisome proliferator activated receptor-c, txo qis hauv cov ntshav thiab cov tshuaj synovial P thiab interleukin-6 qib, inhibition ntawm thromboxane-prostanoid receptor thiab inhibition ntawm acid-sensing ion channels.